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In Vivo 2017

Aureobasidium pullulans-cultured fluid induces IL-18 production, leading to Th1-polarization during influenza A virus infection

Fujikura, Daisuke; Muramatsu, Daisuke; Toyomane, Kochi; Chiba, Satoko; Daito, Takuji; Iwai, Atsushi; Kouwaki, Takahisa; Okamoto, Masaaki; Higashi, Hideaki; Kida, Hiroshi; Oshiumi, Hiroyuki · The Journal of Biochemistry · DOI: 10.1093/jb/mvx062
Research Archive
TL;DR — Key Findings
  • Injected A. pullulans cultured fluid increased IL-18-related responses in mice.
  • The response differed from alum and showed less tested inflammatory-cell recruitment.
  • Animal injection results are not evidence of oral human efficacy.

Abstract

In an influenza A mouse model, intraperitoneal administration of Aureobasidium pullulans cultured fluid increased serum IL-18 and splenic IFN-gamma-producing CD4-positive cells. Cell experiments also showed IL-18 production in a dendritic-cell line and peritoneal cells. Compared with alum, the cultured fluid caused less recruitment of neutrophils and inflammatory macrophages in the tested setting. The material was a cultured fluid containing beta-glucan, the route was injection, and the results do not establish prevention or treatment effects from oral use in humans.

Summary

Summary

What was studied

Aureobasidium pullulans cultured fluid was examined as an immune adjuvant during influenza A infection in mice and in cell assays.

What was observed

Injected cultured fluid increased IL-18 and IFN-gamma-producing CD4-positive cells and produced less local inflammatory-cell recruitment than alum.

How to interpret it

The study used injected cultured fluid in mice. It does not establish oral prevention or treatment effects in humans.

AI-generated summary for accessibility. Always refer to the original paper.

Citation

Fujikura, Daisuke; Muramatsu, Daisuke; Toyomane, Kochi; Chiba, Satoko; Daito, Takuji; Iwai, Atsushi; Kouwaki, Takahisa; Okamoto, Masaaki; Higashi, Hideaki; Kida, Hiroshi; Oshiumi, Hiroyuki. Aureobasidium pullulans-cultured fluid induces IL-18 production, leading to Th1-polarization during influenza A virus infection. The Journal of Biochemistry. 2017. DOI: 10.1093/jb/mvx062.

Study Details
Study Type
In Vivo
Published
2017
Journal
The Journal of Biochemistry
Authors
Fujikura, Daisuke; Muramatsu, Daisuke; Toyomane, Kochi; Chiba, Satoko; Daito, Takuji; Iwai, Atsushi; Kouwaki, Takahisa; Okamoto, Masaaki; Higashi, Hideaki; Kida, Hiroshi; Oshiumi, Hiroyuki
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